_Treatment._--The objects to be attained by treatment will be to remove or mitigate as much as possible the cause inducing the disease; to find a medicine which will lessen the irritability of the heart without weakening it; and, last, to maintain a free urinary secretion and prevent exudation and hypertrophy. So long as there is an increase of temperature, with some degree of scantiness of the urine, it may be safe to believe that there is some degree of inflammatory action existing in the cardiac structures, and as long as any evidence of inflammatory action remains, however moderate in degree, there is a tendency to increase or hypertrophy of the connective tissue of the heart or valves, thereby rendering it almost certain that the structural changes will become permanent unless counteracted by persistent treatment and complete rest.
The tincture of digitalis, in 20-drop doses, repeated every hour, is perhaps the most reliable agent we know to control the irritability of the heart, and this also has a decided influence upon the urinary secretion. After the desired impression upon the heart is obtained the dose may be repeated every two or three hours, or as the case may demand. Tincture of strophanthus, in 2-dram doses, will quiet the tumultuous action of the heart in some cases where the digitalis fails.
Bleeding, blistering, and stimulating applications to the chest should be avoided. They serve to irritate the animal and can do no possible good. Chlorate of pota.s.sium in 2-dram doses may be given in the drinking water every four hours for the first five or six days, and then be superseded by the nitrate of pota.s.sium in half-ounce doses for the following week or until the urinary secretion becomes abnormally profuse. Where the disease is a.s.sociated with rheumatism, 2-dram doses of salicylate of soda may be subst.i.tuted for the chlorate of pota.s.sium.
To guard against chronic induration of the valves, the iodid of pota.s.sium, in 1 to 2 dram doses, should be given early in the disease and may be repeated two or three times a day for several weeks. When chronic effects remain after the acute stage has pa.s.sed this drug becomes indispensable.
When dropsy of the limbs develops, it is due to weakened circulation or functional impairment of the kidneys. When there is much weakness in the action of the heart, or general debility is marked, the iodid of iron, in 1-dram doses, combined with hydrastis, 3 drams, may be given three times a day. a.r.s.enic, in 5-grain doses twice a day, will give excellent results in some cases of weak heart a.s.sociated with difficult breathing.
In all cases absolute rest and warm stabling, with comfortable clothing, become necessary, and freedom from work should be allowed for a long time after all symptoms have disappeared.
PERICARDITIS, OR INFLAMMATION OF THE SAC INCLOSING THE HEART.
_Causes._--Pericarditis may be induced by cold and damp stabling, exposure and fatigue, from wounds caused by broken ribs, etc. Generally, however, it is a.s.sociated with an attack of influenza, rheumatism, pleuritis, etc.
_Symptoms._--Usually the disease manifests itself abruptly by a brief stage of chills coincident with pain in moving, a short painful cough, rapid and short breathing, and high temperature, with a rapid and hard pulse. In the early stages of the disease the pulse is regular in beat; later, when there is much exudation present in the pericardial sac, the heartbeat becomes m.u.f.fled, and may be of a double or rebounding character. By placing the ear against the left side of the chest behind the elbow a rasping sound may be heard, corresponding to the frequency of the heartbeat. This is known as a friction sound. Between the second and fourth days this sound may disappear, due to a distension of the pericardium by an exudate or serous effusion. As soon as this effusion partly fills the pericardium, percussion will reveal an abnormally increased area of dullness over the region of the heart, the heartbeats become less perceptible than in health, and in some cases a splashing or flapping sound may become audible.
If the effusion becomes absorbed, the friction sound usually recurs for a short time; this friction may often be felt by applying the hand to the side of the chest. In a few cases clonic spasms of the muscles of the neck may be present. In acute pericarditis, when the effusion is rapid and excessive, the animal may die in a few days or recovery may begin equally as early. In subacute or in chronic cases the effusion may slowly become augmented until the pressure upon the lungs and interference with the circulation become so great that death will result. Whether the attack is acute, subacute, or chronic, the characteristic symptoms which will guide us to a correct diagnosis are the friction sound, which is always synchronous with the heart"s action, the high temperature with hard, irritable pulse, and, in cases of pericardial effusion, the increased area of dullness over the cardiac region. When the disease is a.s.sociated with influenza or rheumatism, some of the symptoms may be obscure, but a careful examination will reveal sufficient evidence upon which to base a diagnosis. When pericarditis develops as a result of or in connection with pleuritis, the distinction may not be very clearly definable, neither will many recover. When it results from a wound or broken rib, it almost invariably proves fatal.
_Pathology._--Pericarditis may at all times be regarded as a very serious affection. At first we will find an intense injection or acc.u.mulation of blood in the vessels of the pericardium, giving it a red and swollen appearance, during which we have the friction sound. In 24 to 48 hours this engorgement is followed by an exudation of sero-fibrinous fluid, the fibrinous portion of which may soon form a coating over the internal surface of the pericardial sac, and may ultimately form a union of the opposing surfaces. Generally this adhesion will only be found to occupy a portion of the surfaces. As the serous or watery portion of this effusion is absorbed, the distinctness of the friction sound recurs, and may remain perceptible in a varied degree for a long time. When the serous effusion is very great, the pressure exerted upon the heart weakens its action, and may produce death soon; when it is not so great, it may cause dropsies of other portions of the body. When the adhesions of the pericardial sac to the body of the heart are extensive, they generally lead to increased growth, or hypertrophy, of the heart, with or without dilatation of its cavities; when they are but slight, they may not cause any inconvenience.
_Treatment._--In acute or subacute pericarditis the tincture of digitalis may be given in 20 to 30 drop doses every hour until the pulse and temperature become reduced. Whisky or carbonate of ammonia may be given regularly as stimulants. Bandages should be applied to the legs; if the legs are very cold, tincture of capsic.u.m should be first applied; the body should be warmly clothed in blankets, to promote perspiration.
When the suffering from pain is very severe, 10 grains of morphin may be given by the mouth once or twice a day; nitrate of pota.s.sium, half an ounce, in drinking water, every six hours; after the third day, iodid of pota.s.sium, in 2-dram doses, may be subst.i.tuted. Cold packs to the chest in the early stages of the disease may give marked relief, or, late in the disease, smart blisters may be applied to the sides of the chest with benefit. If the disease becomes chronic, iodid of iron and gentian to support the strength will be indicated, but the iodid of pota.s.sium, in 1 or 2 dram doses, two or three times a day, must not be abandoned so long as there is an evidence of effusion or plastic exudate acc.u.mulating in the pericardial sac. Where the effusion is great and threatens the life of the patient, tapping by an expert veterinarian may save the animal.
VALVULAR DISEASE OF THE HEART.
Acute valvular disease can not be distinguished from endocarditis, and chronic valvular affections are generally the result of endocardial inflammation. The valves of the left side are the most subject--the bicuspid or mitral and the aortic or semilunar. The derangement may consist of mere inflammation and swelling, or the edges of the valves may become covered by the organization of the exudation, thus narrowing the pa.s.sage. Valvular obstruction and adhesions may occur or the tendinous cords may be lengthened or shortened, thus obstructing the orifices and permitting the regurgitation of blood. In protected cases the fibrous tissue of the valves may be transformed into fibro-cartilage or bone, or there may be deposits of salts of lime beneath the serous membrance, which may terminate in ulceration, rupture, or fissures.
Sometimes the valves become covered by fibrinous, fleshy, or hard vegetations, or excrescences. In cases of considerable dilatation of the heart there may be atrophy and shrinking of the valves.
_Symptoms._--Valvular disease may be indicated by a venous pulse, jerking pulse, intermittent pulse, irregular pulse; palpitation; constant abnormal fullness of the jugular veins; difficulty of breathing when the animal becomes excited or is urged out of a walk or into a fast trot; attacks of vertigo; congestion of the brain; dropsical swelling of the limbs. A blowing, cooing, or bubbling murmur may sometimes be heard by placing the ear over the heart on the left side of the chest.
Hypertrophy, or dilatation, or both, usually follow valvular disease.
_Treatment._--When the pulse is irregular or irritable, tonics, such as preparations of iron, gentian, and ginger, may be given. When the action of the heart is jerking or violent, 20 to 30 drop doses of tincture of digitalis or of veratrum viride may be given until these symptoms abate.
As the disease nearly always is the result of endocarditis, the iodid of pota.s.sium and general tonics, sometimes stimulants, when general debility supervenes, may be of temporary benefit. Very few animals recover or remain useful for any length of time after once marked organic changes have taken place in the valvular structure of the heart.
ADVENt.i.tIOUS GROWTHS IN THE HEART.
Fibrous, cartilaginous, and bony formations have been observed in some rare instances in the muscular tissue. Isolated calcareous ma.s.ses have sometimes been embedded in the cardiac walls. Fibrinous coagula and polypous concretions may be found in the cavities of the heart. The former consist of coagulated fibrin, separated from the ma.s.s of blood, of a whitish or yellowish white color, translucent, of a jellylike consistence, and having a nucleus in the center. They may slightly adhere to the surface of the cavity, from which they can easily be separated without altering the structure of the endocardium. They probably result from an excess of coagulability of fibrin, which is produced by an organization of the lymph during exudation. They are usually found in the right auricle and ventricle.
Polypous concretions are firmer than in the preceding, more opaque, of a fibrous texture, and may be composed of successive layers. In some instances they are exceedingly minute, while in others they almost fill one or more of the cavities. Their color is usually white, but occasionally red from the presence of blood. They firmly adhere to the endocardium, and when detached from it give it a torn appearance.
Occasionally, a vascular communication seems to exist between them and the substance of the heart. They may be the result of fibrinous exudation from inflammation of the inner surface of the heart or the coagulation of a portion of the blood which afterwards contracts adhesion with the heart. These concretions prove a source of great inconvenience and often danger, no matter how formed. They cause a diminution in the cavity in which they are found, thus narrowing the orifice through which the blood pa.s.ses, or preventing a proper coaptation of the valves, which may produce most serious valvular disease.
_Symptoms._--These are frequently uncertain; they may, however, be suspected when the action of the heart suddenly becomes embarra.s.sed with irregular and confused pulsations, great difficulty of breathing, and the usual signs dependent upon the imperfect arterialization of the blood.
_Treatment._--Stimulants, whisky, or carbonate of ammonia may be of service.
PALPITATION OF THE HEART.
This is a tumultuous and usually irregular beating of the heart. It may be due to a variety of causes, both functional and organic. It may occur as a result of indigestion, fright, increased nervousness, sudden excitement, excessive speeding, etc. (See "Thumps," p. 225.)
_Symptoms._--The heart may act with such violence that each beat may jar the whole body of the animal; very commonly it may be heard at a short distance away from the animal. It can usually be traced very readily to the exciting cause, which we may be able to avoid or overcome in the future and thereby obviate subsequent attacks. Rest, a mild stimulant, or a dose or two of tincture of digitalis or opium will generally give prompt relief. When it is due to organic impairment of the heart it must be regarded as a symptom, not as a matter of primary specific treatment.
SYNCOPE, OR FAINTING.
Actual fainting rarely occurs among horses. It may, however, be induced by a rapid and great loss of blood, pain of great intensity, a mechanical interference with the circulation of the brain, etc.
_Symptoms._--Syncope is characterized by a decrease or temporary suspension of the action of the heart and respiration, with partial or total loss of consciousness. It generally occurs suddenly, though there may be premonitory symptoms, as giddiness, or vertigo, dilated pupil, staggering, blanching of the visible mucous membranes, a rapidly sinking pulse, and dropping to the ground. The pulse is feeble or ceases to beat; the surface of the body turns cold; breathing is scarcely to be perceived, and the animal may be entirely unconscious. This state is uncertain in duration--generally it lasts only a few minutes; the circulation becomes restored, breathing becomes more distinct, and consciousness and muscular strength return. In cases attended with much hemorrhage or organic disease of the heart, the fainting fit may be fatal; otherwise it will prove but a transient occurrence. In paralysis of the heart the symptoms may be exactly similar to syncope. Syncope may be distinguished from apoplexy by the absence of stertorous breathing and lividity of the visible mucous membranes.
_Treatment._--Dash cold water on the head; administer a stimulant--4 ounces of whisky or half an ounce of carbonate of ammonia. Prevent the animal from getting up too soon, or the attack may immediately recur.
Afterwards, if the attack was due to weakness from loss of blood, impoverished blood, or a.s.sociated with debility, general tonics, rest, and nourishing food are indicated.
HYPERTROPHY OF THE HEART, OR CARDIAC ENLARGEMENT.
Hypertrophy of the heart implies augmentation of bulk in its muscular substance, with or without dilatation or contraction of its cavities. It may exist with or without other cardiac affections. In valvular disease or valvular insufficiency hypertrophy frequently results as a consequence of increased demand for propelling power. The difficulties with which it is most frequently connected are dilatation and ossification of the valves. It may also occur in connection with atrophied kidneys, weak heart, etc. It may be caused by an increased determination of blood to the organ or from a latent form of myocarditis, and it may arise from a long-continued increase of action dependent upon nervous disease. All the cavities of the heart may have their walls hypertrophied or the thickening may involve one or more.
While the wall of a ventricle is thickened, its cavity may retain its normal size (simple hypertrophy) or be dilated (eccentric hypertrophy), or it may be contracted (concentric hypertrophy). Hypertrophy of both ventricles increases the length and breadth of the heart. Hypertrophy of the left ventricle alone increases its length; of the right ventricle alone increases its breadth toward the right side. Hypertrophy with dilatation may affect the chambers of the heart conjointly or separately. This form is by far the most frequent variety of cardiac enlargement. When the entire heart is affected, it a.s.sumes a globular appearance, the apex being almost obliterated and situated transversely in the chest. The bulk may become three or four times greater than the average heart.
_Symptoms._--In hypertrophy of the heart, in addition to the usual symptoms manifested in organic diseases of the heart, there is a powerful and heaving impulse at each beat, which may be felt on the left side, often also on the right. These pulsations are regular, and when full and strong at the jaw there is a tendency to active congestion of the capillary vessels, which frequently give rise to local inflammation, active hemorrhage, etc. If the pulse is small and feeble at the jaw, we may conclude that there is some obstacle to the escape of the blood from the left ventricle into the aorta, which has given rise to the hypertrophy. In case of hypertrophy with dilatation, the impulse is not only powerful and heaving, but it is diffused over the whole region of the heart, and the normal sounds of the heart are greatly increased in intensity. Percussion reveals an enlarged area of dullness, while the impulse is usually much stronger than normal.
Dropsy of the pericardium will give the same wide s.p.a.ce of dullness, but the impulse and sound are lessened. An animal with a moderate degree of enlargement may possibly live a number of years and be capable of ordinary work; it depends largely upon concomitant disease. As a rule, an animal affected with hypertrophy of the heart will soon be incapacitated for work, and becomes useless and incurable.
_Treatment._--If the cause can be discovered and is removable, it should be done. The iodid of pota.s.sium, in cases of valvular thickening, may be of some benefit if continued for a sufficient length of time; it may be given in 2-dram doses, twice a day, for a month or more. The tincture of digitalis may be given, in cases where the pulse is weak, in doses of 2 teaspoonfuls three times daily. This remedy should not be continued if the pulse becomes irregular. General tonics, freedom from excitement or fatigue, avoidance of bulky food, good ventilation, etc., are indicated.
DILATATION OF THE HEART.
This is an enlargement, or stretching, of the cavities of the heart, and may be confined to one or extend to all. Two forms of dilatation may be mentioned--simple dilatation, where there is normal thickness of the walls, and pa.s.sive, or attenuated, dilatation, where the walls are simply distended or stretched out without any addition of substance.
_Causes._--Any cause producing constant and excessive exertion of the heart may lead to dilatation. Valvular disease is the most frequent cause. General anemia predisposes to it by producing relaxation of muscular fiber. Changes in the muscular tissue of the heart walls, serous infiltration from pericarditis, myocarditis, fatty degeneration and infiltration, and atrophy of the muscular fibers may all lead to dilatation.
_Symptoms._--The movements of the heart are feeble and prolonged, a disposition to staggering or vertigo, dropsy of the limbs, very pale or very dark-colored membranes, and difficult breathing on the slightest excitement.
_Treatment._--General tonics, rich feed, and rest.
FATTY DEGENERATION OF THE HEART.
Fatty degeneration may involve the whole organ, or may be limited to its walls, or even to circ.u.mscribed patches. The latter is situated at the exterior, and gives it a mottled appearance. When generally involved it is flabby or flaccid, and in extreme cases collapses when emptied or cut. Upon dissection the interior of the ventricles is observed to be covered with buff-colored spots of a singular zigzag form. This appearance may be noticed beneath the pericardium, and pervading the whole thickness of the ventricular walls, and in extreme cases those of the fleshy columns in the interior of the heart. These spots are found to be degenerated muscular fibers and colonies of oil globules. Fatty degeneration is often a.s.sociated with other morbid conditions of the heart, such as obesity, dilatation, rupture, aneurism, etc. It may be connected with fatty diseases of other organs, such as the liver, kidneys, etc. When it exists alone its presence is seldom suspected previous to death. It may be secondary to hypertrophy of the heart, to myocarditis, or to pericarditis. It may be due to deteriorated conditions of the blood in wasting diseases, excessive hemorrhages, etc., or to poisoning with a.r.s.enic and phosphorus.
_Symptoms._--The most prominent symptoms of fatty degeneration are a feeble action of the heart, a remarkably slow pulse, general debility, and attacks of vertigo. It may exist for a long time, but is apt to terminate suddenly in death upon the occurrence of other diseases, surgical operations, etc. It may involve a liability to sudden death from rupture of the ventricular walls.
_Treatment._--Confinement in feed to oats, wheat or rye bran, and timothy hay. Twenty drops of sulphuric acid may be given in drinking water three times a day, and hypophosphite of iron in 2-dram doses, mixed with the feed, twice a day. Other tonics and stimulants as they may be indicated.
RUPTURE OF THE HEART.